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Additional info for Amyloidosis - Mechanisms and Prospects for Therapy
45, No. 1, (January 2006), pp. ] Amyloidotic cardiac involvement has been revealed to trend to be a poor prognostic factor. Heart failure is likely to be directly responsible for death in only a minority of patients, however, patients with heart failure may be complicated by multiple organ failures in later phase of the RA disease course. It seems to be suggested that dysautonomia plays an important role in etiology of heart failure to some extent in patients with AA amyloidosis in addition to direct AA amyloid fibril deposits in situ.
These humoral and cellular inflammatory events interact, with SAA being a key player. RAGE: receptor for advanced glycation end products; FPRL1: formyl peptide receptor-like 1; TLR2, 4: toll-like receptor 2 and 4; CLA-1: CD36 and 38 Amyloidosis – Mechanisms and Prospects for Therapy LIMPII analogous-1, human orthologue of the scavenger receptor class B type I (SR-BI); AGEs: advanced glycation end products. [From Nakamura, T. (2011). Amyloid A amyloidosis secondary to rheumatoid arthritis: pathophysiology and treatment.
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